Decomposition Cycles of a Kaiju/Kaijin Body and the Emergence of Xenonotic Pathogens
The decomposition of a kaiju (Colossal-Mutagenic Monstrous Entity, CMME) or kaijin (Protean-Ontogenic Mutant Monstrous Entity, POMME Unit or naturally occurring mutation) follows a unique biological and biochemical trajectory, distinct from terrestrial organisms. The introduction of Xenologic Induced Teratomutagens (XIT Pathogens) into the environment, and ultimately into human hosts, is a multi-phase process dictated by the decay of xenobiotic organic matter, the metabolic byproducts of its microbiome, and the unique parasitic agents harbored within its tissues. Below is a detailed breakdown of this cycle:
Phase 1: Immediate Post-Mortem (0-12 Hours After Death) – Xenocellular Necrotoxicity
Upon death, a kaiju/kaijin undergoes catastrophic cellular collapse due to the loss of its metabolic bioelectric field—a property theorized to be an endogenous piezoelectric shielding mechanism that prevents the rapid breakdown of its tissues while alive. This results in:
Xenocellular lysis, where hyper-dense alien myofibrils and neural matter begin to degrade, releasing high concentrations of xenoenzymes into the immediate environment.
A spike in biopsyrrhic fluid production, a necrotic ichor composed of highly mutagenic extracellular vesicles that seep into the surrounding ground and waterways.
The release of residual symbiotic xenobacteria—a microbiota uniquely adapted to the kaiju’s internal ecosystem, some of which become opportunistic pathogens in terrestrial hosts.
Spore activation in certain species, particularly those originating from regions with high exposure to cosmic radiation or abyssal environments, such as the Indian Ocean or the Celtic Sea.
At this stage, direct contact with exposed tissue, fluid, or particulate matter carries an extremely high risk of contamination, as proteolytic xenotoxins can penetrate the skin and mucous membranes.
Phase 2: Early Decomposition (12 Hours – 7 Days) – Teratomutagenic Proliferation
The second stage is characterized by biological liquefaction and the first wave of xenonotic pathogen dispersal. This occurs through:
Aerozoonic Dispersal:
The decomposition of xenobiotic tissues releases volatile organic compounds (VOCs) infused with teratomutagenic particulate matter.
The XIT pathogen, which exists as a spore-forming xenoparasite within kaiju tissues, enters its aerobic stage, becoming airborne and dispersing via wind currents.
This results in localized contamination within a several-mile radius, with potential long-distance dispersal in cases of storm systems or coastal winds.
Xenozooinic Vectors (Faunal Contamination):
Scavenger species (birds, insects, small mammals) feeding on kaiju remains become biological carriers, undergoing minor physiological mutations or serving as passive hosts.
Aquatic diffusion occurs as xenoenzymatic compounds leach into groundwater, rivers, and oceanic currents, where filter-feeders and predatory species absorb the pathogen.
Early-stage teratomorphic transformations have been observed in local fauna near Incident Sites (Pacific Northwest, Gulf of Mexico, Atlantic Northeast, Celtic Sea, Indian Ocean).
Groundwater and Soil Contamination:
The breakdown of xenobiotic collagen analogs and hemocyanin-like compounds sterilizes surrounding microbiomes, outcompeting terrestrial bacteria.
Soil mycorrhizal networks become infiltrated with xenocellular growths, resulting in highly mutagenic fungal and plant life.
Waterborne biofilm formation allows for pathogen survival in nutrient-rich aquatic environments, leading to delayed infections through secondary exposure.
Phase 3: Mid-Term Decay (7 Days – 6 Months) – Environmental Integration & Pathogenic Colonization
At this stage, XIT pathogens become entrenched within the local ecosystem, creating a sustained reservoir for future infections. This includes:
Fungal-Xenobacterial Hybridization:
The emergence of xenomycotic biofilms, which spread through spore-based dispersion, embedding XIT pathogens in crops, airborne dust, and decomposing organic matter.
The first documented cases of "night pestilence" outbreaks originate from fungal vector contamination of grain and livestock.
Aquatic & Atmospheric Dissemination:
Persistent planktonic microcolonies allow for long-term viability of XIT spores in deep-sea currents, making coastal fisheries a high-risk vector.
Certain volatile teratotoxins become aerosolized through evaporation cycles, allowing pathogenic particles to bond with water vapor, enabling rainborne infections.
Localized Mutagenesis in Human Populations:
Communities living near kaiju graveyards or contamination zones begin exhibiting increased rates of genetic instability, with reported cases of low-grade teratomorphic anomalies (e.g., polydactyly, hematological disorders, and sporadic cases of mild biomorphic traits).
Individuals consuming contaminated food or water often experience latent viral integration, resulting in symptomless incubation until a triggering event (e.g., extreme stress, secondary infection, or exposure to another pathogen).
Phase 4: Late-Stage Decomposition (6 Months – 10+ Years) – Human Infection & Night Pestilence Outbreaks
By this point, XIT pathogens have fully established themselves in the environment. The most common human infection vectors are:
1. Direct Contact with Infected Soil & Water
Individuals working in agriculture, fishing, or forestry in contaminated zones often experience low-grade symptoms of night pestilence, with delayed onset mutations.
Certain traditional medicine practices, including the use of kaiju remains as fertilizers or medicinal powders, have led to cultural transmission events.
2. Inhalation of Airborne Spores
Seasonal outbreaks coincide with temperature shifts, wildfires, or storms, which reactivate dormant XIT spores in soil and vegetation.
Urban populations downwind from incident sites are at risk due to xenonotic microspores accumulating in air filters, textiles, and ventilation systems.
3. Secondary Transmission via Kaijin Bites or Bloodborne Exposure
Once the XIT pathogen is active within a human host, it spreads through direct fluid exchange, including bites, scratches, and transfusions.
POMME Units, designed as controlled kaijin, often face stigma due to their perceived role as carriers, despite their modified immune resistance to full transformation.
Conclusion: The Xenonotic Cycle of Night Pestilence
The decomposition of kaiju/kaijin bodies is not merely an ecological event—it is a biological weapon left in the wake of every Incident. Unlike terrestrial diseases, which burn out over time, XIT pathogens integrate into their environment, allowing for cyclical resurgences. Unchecked contamination zones serve as permanent reservoirs for future outbreaks, making containment, eradication, and strategic cleansing essential to preventing global mutations.
Only through rigorous monitoring, biological countermeasures, and controlled POMME deployment can the threat of night pestilence be mitigated. But as history has shown, eradication is impossible—only delay.
